乙型流感病毒IgA ELISA免疫診斷試劑盒

廣州健侖生物科技有限公司

廣州健侖長(zhǎng)期供應(yīng)各種流感檢測(cè)試劑，包括進(jìn)口和國(guó)產(chǎn)的品牌，主要包括日本富士瑞必歐、日本生研、美國(guó)BD、美國(guó)NovaBios、美國(guó)binaxNOW、凱必利、廣州創(chuàng)侖等主流品牌。

?乙型流感病毒IgA ELISA免疫診斷試劑盒

我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團(tuán)菌、化妝品檢測(cè)、食品安全檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

歡迎咨詢

歡迎咨詢2042552662

想了解更多的產(chǎn)品及服務(wù)請(qǐng)掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】    楊永漢

【】 
【騰訊  】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103室

通過轉(zhuǎn)換儲(chǔ)能的白色脂肪成為燃燒卡路里的棕色或米色脂肪，Metrnl增加能量消耗，提高肥胖、糖尿病小鼠的代謝健康。這種激素，有可能適合作為肥胖和糖尿病的治療新靶標(biāo)，Metrnl是通過激活免疫分子白細(xì)胞介素-4和白細(xì)胞介素13，以及位于脂肪組織的免疫細(xì)胞即巨噬細(xì)胞發(fā)揮這些積極的效果。
發(fā)表在同一雜志上的另一項(xiàng)研究，進(jìn)一步揭示了免疫反應(yīng)和肥胖的相關(guān)問題，舊金山加州大學(xué)Ajay Chawlazui近的研究探究了在應(yīng)對(duì)寒冷環(huán)境中，免疫系統(tǒng)激活棕色脂肪?，F(xiàn)在Chawla和他的團(tuán)隊(duì)發(fā)現(xiàn)了小鼠體內(nèi)米色脂肪活化的基本“線路”。他們發(fā)現(xiàn)，interleukin-4/13信號(hào)激活白色脂肪組織中的巨噬細(xì)胞，導(dǎo)致轉(zhuǎn)換白色脂肪成為米色脂肪所需的神經(jīng)系統(tǒng)分子的生成。這些發(fā)現(xiàn)揭示了重要的新見解，即免疫系統(tǒng)和神經(jīng)系統(tǒng)如何協(xié)同工作來刺激米色脂肪的形成。
此外，研究人員發(fā)現(xiàn)，這種免疫通路信號(hào)受損的小鼠產(chǎn)生較少的米色脂肪，具有較低的能量消耗，并且在寒冷的環(huán)境中，與正常小鼠相比無法維持自己的體溫。與此相反，用白細(xì)胞介素-4處理小鼠能增加米色脂肪的形成，降低體重，改善肥胖小鼠代謝健康。

Metrnl increases energy expenditure and improves the metabolic health of obese, diabetic mice by converting stored energy white fat to brown or beige fat that burns calories. This hormone, potentially suitable as a novel target for the treatment of obesity and diabetes, Metrnl exerts these positive effects by activating the immune molecules interleukin-4 and interleukin-13, as well as the immune cells located in adipose tissue, macrophages.
Another study, published in the same journal, further uncovered questions about the immune response and obesity. A recent study by Ajay Chawla at the University of California, San Francisco, explored the activation of brown fat by the immune system in response to cold conditions. Now Chawla and his team found the basic "line" of beige fat activation in mice. They found that interleukin-4/13 signaling activates macrophages in white adipose tissue, resulting in the production of neuronal molecules needed to convert white fats to beige fats. These findings reveal important new insights on how the immune system and the nervous system work together to stimulate the formation of beige fats.
In addition, the researchers found that mice with impaired immune signaling generated less beige fat, had lower energy expenditure, and were unable to maintain their body temperature in cold conditions compared to normal mice. In contrast, treatment of mice with interleukin-4 increased the formation of beige fat, decreased body weight, and improved the metabolic health of obese mice.